Nasal Surgery Side Effects & Risks – Injuries from Septoplasty & Turbinate Reduction – Empty Nose Syndrome

In this blog post, you will learn about the injuries that the healthcare system doesn’t want to mention in connection with nasal surgery on the turbinates and septum. You can access the information either by reading the blog post in text form or by watching the YouTube video below. Alternatively, you can listen to the information in podcast format. Below, you’ll find several podcast platforms to choose from—please note that the same information is available on all of them.

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Welcome. In blog post, we will uncover the harsh truth about modern nasal surgery performed for nasal congestion—the truth that today’s surgeons do not want you to know about before agreeing to the procedure. The reality is that these surgeries, even when technically successful, can severely disrupt the autonomic nervous system—not just locally in the nose, but throughout the entire body. For many individuals, this leads to life-altering consequences, shattering their lives completely.

Many patients suffer lifelong and severe breathing problems as a result of these procedures, often in the form of paradoxical obstruction—a condition where, due to nerve damage and loss from the surgery, the nose will always feel blocked, even when it is physically open. This condition triggers an overactivation of the sympathetic nervous system, causing chronic stress and severe sleep disturbances.

Additionally, these surgeries frequently lead to complications involving the remaining nasal mucosa. The problem is not only what is removed but also what is left behind. After an aggressive turbinate reduction, the remaining mucosa often dries out, leading to degeneration. A common consequence is recurring nasal infections, which further deteriorate the mucosa. Over time, the mucosa becomes so dry that affected individuals can no longer tolerate normal environments without extreme discomfort and nasal pain.

The situation is made even worse by the fact that surgeons systematically withhold information about these risks and complications before the surgery. The only risks typically mentioned are bleeding and infection related to the procedure itself. All long-term, irreversible consequences—many of which are almost guaranteed—are deliberately omitted to avoid scaring patients away.

Before we dive into the full presentation, I want to emphasize that the reason behind this video is my own irreversible iatrogenic injury to my nasal mucosa and function following a routine, modern nasal surgery involving turbinate and septum reduction. After realizing the extent of the damage, I have spent thousands of hours researching this issue. In this video, I will present my findings to protect others from ending up in the same devastating situation—one that has completely destroyed my life.

Empty Nose syndrome / Iatrogenic Nasal Dysfunction

The condition I am about to describe is referred to in the medical literature as Empty Nose Syndrome (ENS). However, I personally prefer to call it Iatrogenic Nasal Dysfunction, as the term Empty Nose Syndrome falsely implies that the nose must be completely void of structures for someone to develop the condition. This is incorrect. A person who has undergone turbinate or septal surgery can still develop the condition, even if a significant portion of their turbinates remain intact. A more accurate term would be Iatrogenic Nasal Dysfunction, since iatrogenic refers to harm caused by medical treatment. This term better describes the root cause of the condition—a dysfunction of the nose and its structures resulting from a medically induced injury, specifically nasal surgery.

For simplicity, we will continue referring to the condition as Empty Nose Syndrome in this video, using the abbreviation ENS moving forward.

ENS is a severe condition that affects not only the nose but the entire body—both physically and mentally. The condition is highly likely to occur if a surgeon performs an aggressive turbinate or septal reduction, damaging, burning, or removing too many sensory neurons from the nasal mucosa. ENS is therefore primarily a nerve-related disorder, though other contributing factors exist, which we will explore later.

ENS encompasses several different issues, the most defining being paradoxical obstruction. This occurs when, due to nerve damage and sensory loss, the nose feels congested despite being physically open. Patients with this condition can no longer perceive airflow in their nose, meaning their brain no longer registers nasal breathing. This results in a constant sensation of air hunger and breathlessness, which is mentally and physically exhausting since the sensation is often persistent.

Other common issues in ENS include a lack of normal airway resistance, leading to shallow and rapid breathing. This not only causes mental distress but can also negatively impact oxygenation on a physiological level. You can learn more about this in my previous videos, which are linked in the description below.

ENS also includes complications related to the remaining mucosa, such as chronic dryness, recurrent nasal infections, pain, and discomfort from dry air. Many ENS sufferers experience extreme sensitivity and pain triggered by odors, airborne particles, and smoke. As a result, many are forced to rely on humidifiers 24/7 to compensate for their nose’s inability to produce adequate secretions. Additionally, ENS often disrupts the nasal cycle, causing either an abnormal, painful cycle or a complete absence of one. In many cases, both nostrils remain constantly wide open, further worsening mucosal dryness.

Since ENS frequently leads to severe sleep disturbances and chronic stress, it also accelerates aging and increases the risk of secondary health conditions. If you want to read about all known ENS-related complications, please refer to the link below.


After researching this condition for several years, I can say that affected individuals suffer immensely. Many are unable to work, and many have lost both friends and partners. It is also not uncommon for severely affected individuals to take their own lives. During the years I have studied this condition, I have personally been informed of about ten people who ended their lives due to their suffering—and of course, I am not aware of all cases. Unfortunately, in most cases, the condition is irreversible, which is why the best treatment is prevention: never developing it in the first place. In other words, one should avoid nasal surgery and instead treat nasal congestion through natural methods. This is because Empty Nose Syndrome (ENS) can only develop as a result of nasal surgery. While it can also occur due to a prolonged and severe infection of the nasal mucosa, this is typically secondary to the dryness and immunosuppressive effects caused by the original surgery.

What many people struggle to understand is that ENS can either begin immediately after nasal surgery or develop gradually over time. It is not uncommon for affected individuals to experience severe symptoms only years after their initial surgery, making it difficult for them to link their problems to the procedure. This happens because the surgery itself damages the nose’s ability to keep the mucosa moist and healthy, leading to its gradual degeneration due to dryness. The timeline for such degeneration—or whether it occurs at all—depends on the extent of tissue damage and whether the nasal cycle is still functional, allowing each nostril to rest and recover in shifts. It also depends on the environment in which one lives—those in dry and warm climates may experience degeneration more rapidly.

Additionally, surgery on the turbinates and nasal septum often results in weakened immunity within the nose. This is because the procedure removes or damages nasal tissue that contains goblet cells, which produce mucus rich in antibacterial substances such as lysozyme, lactoferrin, nitric oxide, and antibodies—all of which have strong antibacterial and antiviral properties. As a result, it is common for affected individuals to eventually develop infections in the nasal mucosa, further damaging it and worsening airflow sensation and dryness, as even more goblet cells are destroyed.

After researching this condition for over four years and speaking with and reading the testimonies of hundreds of patients, I can also conclude that ENS almost always leads to severe sleep disturbances. Many affected individuals struggle to sleep more than one to two hours at a time before waking up gasping for air, even when taking sleeping pills. These medications frequently cause anticholinergic side effects, which exacerbate the pre-existing nasal dryness caused by the surgery. The anticholinergic effects also lead to additional problems, such as dry eyes, dry mucous membranes, dry mouth, and gastrointestinal issues due to reduced mucus production in the intestines.


Surgeons Blaming Poor Healing After Surgery = Gaslighting

What makes this condition unique is that it can only develop as a direct result of surgery. Some surgeons try to claim that poor healing is to blame. However, this is a lie designed to shift responsibility from the surgeon to the patient. On the contrary, the surgery itself is 100% responsible for the damage and the condition. The more aggressively a surgeon cuts away, trims, or burns nasal tissue during the procedure, the more severe the complications the patient will have to endure—either immediately or years later when the remaining mucosa has degenerated.

We must remember that during the surgery, the surgeon is cutting, trimming, or burning away sensory neurons in the nasal mucosa. In doing so, they destroy the nerves responsible for detecting airflow in the nose, which is essential for comfortable and relaxed breathing. When the patient later returns with problems, the surgeon often claims that poor healing is to blame. This is as absurd as saying that a patient’s inability to walk is due to poor healing after having their left leg amputated. This is medical gaslighting of the highest order—just like claiming that the turbinates are still present after a radiofrequency treatment and that it is impossible for the patient to have developed ENS as a result.

ENT Departments – How They Operate
Now, let’s move on to the next topic and examine how ENT (Ear, Nose, and Throat) departments function. Understanding this is crucial for any patient considering surgery. When a patient visits their local ENT specialist, they are not typically seeing a general physician who is primarily interested in investigating the root cause of their nasal congestion on a systemic level. Instead, the first person they meet is often a surgeon. These surgeons are employed mainly to perform nasal surgeries—whether or not they are truly necessary.

In fact, a large part of ENT department operations revolves around performing surgeries; the entire system is heavily dependent on this. To maintain their annual budget from the government, a certain number of nasal congestion surgeries must be performed each year. If fewer of these procedures are carried out, the department’s budget will decrease over time, eventually leading to job losses among surgeons. Additionally, many surgeons now work as consultants, selling their services to hospitals—meaning they have a personal financial incentive to perform as many and as extensive surgeries as possible.

It’s also important to understand that the highest-ranking individuals within ENT departments are typically those with the most education. Since surgeons generally have the most extensive medical training, they often become department heads, further steering the department’s focus toward more surgeries. As a result, the structure of the ENT field is unfortunately built in a way that is detrimental to individual patients. Because the goal is to perform a high number of surgeries, ENT specialists often fail to investigate the actual cause of nasal congestion before recommending surgery. A proper investigation would likely reveal that at least 90% of cases could be resolved naturally, without surgery.

There are many systemic causes of nasal congestion that can be treated without surgery. While this video is not intended to cover all of them, it is essential for patients to research and gather information before considering an operation. However, before we move on, let’s briefly mention some of the most common non-surgical causes of nasal congestion.

Natural Causes of Nasal Congestion
Some natural causes of nasal congestion include allergies (IgE- or IgA-mediated), food intolerances with IgG activation, and inflammatory conditions in the body such as autoimmune diseases. Mold, dust, pollen, or other irritating chemical substances in your environment can also contribute. Additionally, hormonal changes in the body can cause nasal congestion, particularly in women during pregnancy when estrogen levels rise. Another cause can be a deficiency of the DAO enzyme, which breaks down histamine—a compound that can trigger nasal congestion.

Long-term use of nasal sprays is another major cause. Nasal sprays themselves irritate the mucosa, and over time, their decongestant effect can lead to an increase in blood vessels in the mucosa to compensate for a chronic lack of blood supply. Moreover, the preservatives used in nasal sprays have been shown in laboratory studies to have cell-damaging effects, which can irritate and damage the nasal mucosa.

Many people are unaware that certain foods and beverages can cause nasal congestion. Examples include foods and drinks high in histamines, sulfites, or tannins. In general, aged foods tend to contain high histamine levels—such as aged cheese, canned tuna, fermented dairy products, non-fresh seafood, canned foods, and processed meats. When it comes to drinks, wine and beer are well known for causing nasal congestion. Additionally, processed foods, unhealthy oils, and fried foods like French fries can trigger congestion indirectly by promoting systemic inflammation. The same happens when consuming foods that trigger an immune response due to the presence of specific antibodies. Even healthy foods can cause issues if a person has an individual intolerance to them.

It is therefore recommended to conduct an IgG food intolerance test privately, as this is not typically offered in standard healthcare. Faster allergic reactions (IgE- or IgA-mediated) can be tested through regular healthcare services, but these are usually easier to identify since they occur shortly after consuming the triggering food. IgG reactions, on the other hand, can manifest hours or even days after exposure, making testing a more reliable approach for detection.

As you can see, there are many natural causes of nasal congestion that can be addressed without surgery. However, ENT (ear, nose, and throat) clinics are primarily focused on nasal surgeries, so they do not conduct such tests. If they did, many patients would likely be removed from the surgical queue—something that surgeons naturally want to avoid. Instead, some questionable tactics are used to promote surgery, which we will examine later.

Regardless, the prevailing message from healthcare providers is that the only risks associated with nasal surgeries are infection and bleeding. Unfortunately, this is far from the truth. The reality is that these procedures almost guarantee damage and complications rather than just risks. The real question is not whether you will experience damage, but rather the extent of the damage and how much it will affect you. These consequences are what we will now explore—since the medical field deliberately avoids informing you about them

Why Is It Difficult to Breathe Through the Mouth When the Nose Is Blocked?

Let’s begin. If you are reading this, you have likely experienced nasal congestion at some point and found it uncomfortable. But have you ever wondered why it feels so difficult? Have you ever questioned why complete nasal blockage is stressful and exhausting? Have you ever thought about why it is hard to sleep with a congested nose?

After all, you can always breathe through your mouth, which allows even more airflow than your nose ever could. So why does mouth breathing feel so uncomfortable?

This is where one of the biggest misconceptions in healthcare becomes apparent. The fact is that nasal breathing stimulates the parasympathetic nervous system—the part of the autonomic nervous system responsible for relaxation, recovery, digestion, and healing. Mouth breathing, on the other hand, activates the sympathetic nervous system, which is dominant during physical activity, movement, and stress responses.

When we breathe through the nose, nerve endings in the mucosa—called receptors or sensory neurons—are stimulated as air flows along the surface. These receptors act like sensors in a self-driving car, collecting data that helps the body's control system make appropriate responses. They transmit information to the brain’s respiratory center via the trigeminal nerve (cranial nerve V), allowing the brain to process breathing-related stimuli and regulate respiratory function accordingly.

There are many types of receptors in the nasal mucosa, each responsible for detecting different sensations and conditions. Without going into excessive detail, it is essential to know that the nasal mucosa has a high concentration of parasympathetic nerve fibers connected to the brain via the trigeminal nerve. These fibers are sensitive to various stimuli, including airflow and temperature changes. When these fibers are stimulated by nasal breathing, they trigger a parasympathetic response that promotes relaxation and well-being.

In this sense, nasal breathing can be seen as a continuous soothing massage for the brain and the parasympathetic nervous system. It is no coincidence that most meditation techniques emphasize slow, deep breathing through the nose. Nasal breathing helps maintain dominance of the parasympathetic nervous system, supporting healing, recovery, digestion, sleep, and relaxation. Numerous studies have also shown that nasal breathing enhances cognitive function, improves decision-making, and boosts memory—all due to the mechanical stimulation of these parasympathetic nerve fibers as air flows through the nasal mucosa.

Before undergoing nasal surgery, it is crucial to understand that such procedures do not just affect the nose itself but also impact the entire autonomic nervous system. In other words, nasal surgery is indirectly a procedure on the brain and nervous system—something surgeons conveniently avoid mentioning.


What happens to the nasal mucosa if you undergo surgery on the nasal septum or turbinates?

Yes, even if surgeons don’t say it, all surgery on the turbinates is essentially aimed at destroying them. Either they are partially or completely cut away, or they are burned using various heat-based methods that destroy the tissue and cause it to shrink.

I have previously made a YouTube video explaining how heat-based methods destroy the mucosa, so I won’t go into too much detail here, but I refer to my earlier video. In short, the sensory neurons that detect airflow are located on the surface of the turbinates. When these structures are burned, the surface where these receptors are located is destroyed as well. The tissue is transformed into scar tissue, and the affected individual loses all sensation of airflow in that area. The same thing happens if the turbinates are cut away—this guarantees that all receptor types in that area are permanently lost. Deep inside the nose, there is still a nerve connection to the brain via the fifth cranial nerve, but the outer nerve fibers that once detected airflow on the turbinates are gone. So even though the nerve connections to the brain remain, the brain can no longer receive information about airflow from this now-amputated region.

This raises the question: how much tissue can be burned or amputated before so many nerve fibers are lost that the brain can no longer receive a satisfactory signal that breathing is occurring through the nose? The patient will only find out the answer after the surgery. No surgeon can guarantee this in advance or determine where your personal threshold lies.

Additionally, it’s important to understand that the turbinates have other functions that are lost when they are burned or surgically removed. The surface of the turbinates contains goblet cells that produce mucus containing antibacterial substances. This mucus keeps the mucosa moist and healthy while protecting it from infections. If the turbinates are removed or burned, this function is also lost. The turbinates also have cilia, tiny hair-like structures that help clear the mucosa of dust, particles, bacteria, and old mucus by rhythmically moving it backward toward the throat, where it is swallowed and neutralized by stomach acid. This function is also destroyed when the turbinates are removed or burned.

Beyond this, the turbinates have several physical functions. One of these is increasing the mucosal surface area in the nose, which enhances the perception of airflow and stimulates parasympathetic nerve fibers, promoting a calming and relaxing effect on the nervous system. A larger mucosal surface also means more goblet cells, which produce mucus to keep the mucosa moist. Moreover, the turbinates play a role in the nasal cycle, which alternates airflow between the left and right nostrils, allowing one side to rest while the other handles breathing. This also maintains consistent breathing resistance despite the shift between sides.

As I have described in some of my previous videos, several research studies have confirmed that mechanical airflow stimulation of sensory nerve fibers in the left and right nostrils affects the brain differently. For example, left-nostril breathing has been shown to stimulate the right hemisphere of the brain, which is more involved in emotional and creative processes. It has also been found that left-nostril breathing can increase activity in the limbic system, including the amygdala, which influences emotions and stress responses. Meanwhile, stimulation of sensory nerve fibers in the right nostril activates the left hemisphere, which is more associated with analytical thinking, language, and logical processing. Right-nostril breathing has also been found to influence activity in the prefrontal cortex and temporoparietal regions, which affect concentration, attention, and cognitive functions.

All of this is directly linked to the fact that sensory neurons in the left and right nostrils have different connections to the brain. Hopefully, you can now better understand that nasal surgery is not just a local procedure but also an operation on the brain and nervous system. In fact, nasal surgery can even alter your personality and cognitive functions if these sensory neurons are destroyed. After thousands of hours of research, I can conclude that many people with Empty Nose Syndrome (ENS) experience cognitive difficulties such as issues with attention, concentration, and memory.

So, to answer the initial question—why is it difficult to breathe through the mouth when your nose is blocked? The answer is that the sensory neurons in the nose, which are connected to the parasympathetic nervous system, are not being mechanically stimulated by airflow along the mucosa in this blocked state. The brain is thus deprived of this neural stimulation and signaling, leading to sensations of breathlessness and tension. In such a situation, the only thing a person can think about is using nasal spray to open the nose. Only then can they feel comfortable and at ease again.

Now, knowing this, one should ask: is it really a good idea to operate on the nose and surgically remove or burn away these sensory neurons? Of course not. When enough of these neurons are destroyed, the individual will always feel congested, even though their nose is now physically open.


Laminar Airflow in the Nose Transitions to Turbulent Flow

Furthermore, it should be noted that the nasal turbinates, through their shape, size, and placement, transform laminar airflow upon inhalation into turbulent airflow deeper in the nasal cavity after the air encounters the turbinates. This turbulent flow better distributes odor molecules for improved olfaction while also enhancing airflow perception and prolonging the contact time between inhaled air and the mucosa. This allows for greater humidification and warming of the inhaled air, which is crucial for proper gas exchange in the lungs. The turbinates also ensure that the air is warm and humid before reaching the back of the nose and throat. When the turbinates are significantly reduced or removed, many individuals experience dryness and pain at the back of the nose and throat. Additionally, the turbinates reduce the open diameter of the nasal cavity, creating a Venturi effect that increases the velocity of inhaled airflow. To describe this effect: air enters the nose through the external nostril, where the diameter is large, and airflow velocity is relatively low. When the air later reaches the front part of the turbinates, where the nasal cavity's diameter is smaller due to the turbinates, the airflow velocity increases. This increased velocity results in greater activation of nerve fibers on the mucosal surface, including the turbinates themselves, the mucosa covering the nasal septum, the nasal floor, and the lateral wall. This leads to an enhanced sensation of airflow in the nose and stronger activation of sensory neurons. If the turbinates are removed or significantly reduced, the velocity of the air decreases significantly. Turbulent airflow now transitions into laminar airflow, reducing the perception of airflow even on the remaining mucosa that was not removed. This can contribute to the sensation of breathlessness, which is well documented in Empty Nose Syndrome (ENS).

Nasal Surgery and Its Impact on Airflow

It is also important to note that each nostril contains three turbinates: the inferior, middle, and superior turbinates. Air is distributed around and between these turbinates, and if, for example, the inferior turbinate is removed or significantly reduced, air will primarily take the easiest path to the lungs—along the nasal floor. Airflow towards the upper part of the nose, including the middle turbinate, the septal swell body, and the superior turbinate, will now decrease and mainly be of a laminar nature. This leads to a reduced perception of airflow in these areas as well. Reducing or removing the inferior turbinates, which is common in nasal surgery to treat nasal congestion, can therefore result in a decreased perception of airflow in other parts of the nose. This can exacerbate the effects of nerve loss from the cauterized or amputated turbinates, causing even greater problems with sensory loss and breathlessness.

We have now discussed turbinate surgery and how it can result in breathing difficulties such as breathlessness and disrupted nasal airflow patterns. What we have not yet discussed is how septal surgery can have similar consequences. Many mistakenly believe that only turbinate surgery can lead to breathing issues, dryness, and ENS, but the fact is that any nasal surgery affecting the mucosa can impair the ability to perceive airflow in the nose. This impairment is the primary cause of the breathlessness associated with ENS. The question is how much tissue can be damaged before the problems become so severe that the brain no longer receives sufficient information that breathing is occurring through the nose. One thing is certain: the more mucosa that is damaged—whether it be the turbinates, the nasal septum, or other nasal mucosa—the greater the likelihood of nasal dryness and loss of sensation, and thus the higher the risk of developing ENS after surgery. A combined surgery involving both the turbinates and the nasal septum significantly increases the likelihood of experiencing issues with dryness, sensory loss, and chronic breathlessness compared to surgery involving only the nasal septum or only the turbinates.

It should also be noted that septal surgery is very often combined with partial removal of the inferior turbinates, frequently without informing the patient. I personally had the majority of my inferior turbinates removed during a septoplasty without my knowledge. Neither the surgeon nor anyone else informed me that a vital organ would be removed in conjunction with the septoplasty. I have heard similar stories from many other affected patients. It was not until four years later, during a private radiological examination, that I realized that most of my inferior turbinates were missing.

Now, let us proceed to examine how septoplasty can cause ENS-related issues. Firstly, it is essential to understand that having a deviated nasal septum is normal. Eighty percent of the world’s population has a more or less deviated septum, and this is a natural variation. Most people are born this way, and when born with a deviated septum, the turbinates adjust their shape and size according to the available space in each nostril. They become smaller on the concave side and larger on the convex side, which usually results in an equally open space for airflow in both the right and left nostrils. In most cases, a deviated septum is not the true cause of nasal congestion, but surgeons often do not explain this because they want to perform the surgery.


Risks and Complications of Nasal Septum Surgery

Yes, in my previous lecture, I explained how such a surgery can damage the mucosa. If you want to see this in detail, I refer you to that video, and I will leave a link in the description. However, I want to briefly mention a few things here that are very likely to occur.

First, it is common for a nasal septum operation to alter the external shape of the nose, and this is usually not for the better. The nose often becomes crooked and uneven, and it is very common to develop nasal valve collapse. Besides being aesthetically unpleasing when one or both nostrils collapse, this usually has a very negative effect on breathing and the perception of airflow. When the roof of the nose collapses, airflow to the upper part of the nose is completely or partially blocked. This makes it more difficult for air to reach the septal swell body, where many sensory neurons are located. It also makes it harder for air to reach the middle and upper turbinates and the upper part of the nose. This significantly reduces the sensation of having an open nose and experiencing comfortable breathing, which in itself can cause issues such as shortness of breath—even if no tissue has been removed or destroyed. It should also be noted that surgery to correct nasal valve collapse is extensive and highly invasive. I have personally heard many accounts of such procedures resulting in even greater problems than the original issues.


Septoplasty Causes Mucosal Damage – The Mucosa Tears Apart!

In addition to this, it is important to know that performing surgery on the nasal septum requires detaching the mucosa from the cartilage wall of the septum. This must be done in such a way that the connective tissue layer (perichondrium), which lies between the cartilage and the mucosa, is separated as a single unit along with the mucosa. This is crucial because the mucosa itself is delicate and can easily tear. Many surgeons neglect this and fail to separate them together in one piece, increasing the risk of the mucosa tearing apart. Even if the surgeon successfully detaches both the mucosa and perichondrium together, many surgeons are too aggressive and, at some point during the procedure, end up tearing the thin mucosa in multiple places. Such damage is irreversible.

When the mucosa tears, nerves and blood vessels are severed. The mucosa then loses sensation partially or entirely, and the large blood vessels that wind through it are cut and will not reconnect. The only blood vessels remaining in the septal mucosa are then the small capillaries. This makes the mucosa paper-thin and fragile. Since the larger blood vessels are now severed and partially or completely gone, the mucosa becomes dry and can no longer swell and contract in sync with the turbinates. As a result, the nose remains more open, which negatively affects the ability to sense airflow. This issue can be further exacerbated if the surgeon removes cartilage from the septum, making the nose even more open. From earlier in this video, we already know that the nose should not be too open, as this reduces airflow velocity to the point where the remaining functional sensory neurons struggle to detect that air is flowing along the mucosa. It should also be noted that reduced blood flow to and within the septal mucosa due to surgery leads to a drier mucosa.


Destruction of the Septal Swell Body During Septoplasty

Furthermore, it is common for this type of surgery to damage the area on the upper part of the nasal septum known as the septal swell body. This area, located in the upper part of the nose in front of the attachment of the middle turbinate, has a function similar to that of the turbinates. Sometimes referred to as the "fourth turbinate," it contains a high density of sensory neurons that are essential for the nose to detect airflow, providing an individual with a comfortable breathing sensation. The area also contains a large number of goblet cells that produce mucus to keep the mucosa moist, healthy, and free from infections. Surgery on the nasal septum often completely or partially destroys this area. The likelihood of damage increases if the mucosa is detached far back in the nose near this area or if the surgeon tears off the blood vessels and nerves connecting to the swell body during the procedure.

Finally, it should also be noted that the connective tissue layer called perichondrium, which lies between the cartilage wall and the mucosa, is vital for the cartilage cells of the nasal septum. The perichondrium supplies the cartilage cells with blood and nutrients. If this layer is stripped away on both sides of the septum, the cartilage cells will die. When this happens, a hole in the septum will form some time after the surgery. The size of the hole can vary depending on the affected area. In the worst-case scenario, the hole can become so large that both nostrils essentially merge into one. This weakens the cartilage wall and often leads to nasal valve collapse. As a result, airflow in the nose is disrupted, and it may also cause whistling or disturbing noises when breathing through the nose.

As you can see, nasal septum surgery can cause significant complications that may result in lifelong and troublesome issues—none of which your surgeon will tell you about. The only risks they will mention before surgery are those related to bleeding and infection.

It would be one thing if healthcare providers honestly informed patients about the consequences of this type of surgery, allowing them to make an informed decision on whether to take the risk or not. However, when this information is deliberately withheld to facilitate more surgeries or to avoid alarming patients and dealing with follow-up questions, it amounts to nothing less than medical malpractice. The real question is how society and politicians can allow the consequences of these surgeries to be concealed from patients. Shouldn't it be reasonable for a patient to receive complete information before undergoing a procedure so that they know what they are getting into?

The image below presents a summary of some follow-up studies conducted on nasal surgeries performed to relieve nasal congestion. This summary was compiled by Dr. Steven Houser, and the article is titled "The Diagnosis and Management of Empty Nose Syndrome." I have linked the article below in the video description for those who wish to read it in full.

Now, let us examine one of the studies mentioned in the article, namely "Moore et al., 1985." The study shows that patients who had their inferior turbinates completely removed experienced severe issues as early as one year post-surgery. The follow-up period spanned between two and seven years after surgery. The results revealed that eighty-nine percent of all patients had developed problems with dryness and crusting in the remaining nasal mucosa. Additionally, sixty-six percent had developed atrophic rhinitis due to the surgery—a degenerative condition where the mucosa deteriorates due to dryness. Furthermore, twenty-two percent had developed ozena, a form of atrophic rhinitis characterized by foul-smelling crust formation in the nose, typically caused by the bacterium Klebsiella.

To summarize, approximately nine out of ten operated patients suffered from dryness and crusting in the remaining mucosa, around seven out of ten developed atrophic rhinitis, and about two out of ten developed ozena. Given these statistics, how can it be acceptable for healthcare providers to perform these surgeries at all? More importantly, how can they mislead patients into undergoing these procedures without informing them of the consequences, when the likelihood of experiencing these issues ranges between twenty and ninety percent? This modern-day lobotomy in healthcare must come to an end, so do not be deceived into having your turbinates removed. They are essential breathing structures, and you need them. All mammals have turbinates. The only mammal that believes they can be removed is humans.

Unnecessary Surgery on the Nasal Septum and Turbinates

Finally, let us examine a method that surgeons use as a sales pitch to persuade unsuspecting individuals into surgery. They claim that nasal congestion occurs because the turbinates have grown too large, necessitating their removal or cauterization to restore normal breathing. But consider this: How many other organs in the body continue to grow uncontrollably large? Do you know of any? No? That is because the body does not work that way.

Let us look at how the process unfolds. The first step when evaluating a new patient is to send them for an X-ray. Often, the X-ray will reveal that the nasal septum is deviated and, therefore, must be surgically corrected. As you may know, eighty percent of all people have some degree of a deviated nasal septum. However, since this information is withheld from patients, surgeons can always claim that their nasal congestion is caused by the deviation and that surgery is necessary. If this claim does not succeed, and they still wish to push for surgery, they use another tactic. They argue, based on the same X-ray, that one or both nasal turbinates are too large and must be reduced.

This reasoning suggests that nasal structures, for some unknown reason, continue to grow uncontrollably until they must be cut away or burned down. However, that is not how the body functions. The organs of the body have predetermined sizes; they do not grow indefinitely. What is unique about the turbinates is that they are densely packed with blood vessels that can expand or contract in response to signals from the autonomic nervous system. This process is part of what is known as the nasal cycle. The nasal cycle causes the turbinates and mucosa on one side of the nose to swell while the opposite side contracts, alternating every few hours. Thus, it is impossible to take a single X-ray image in a fraction of a second and claim that one side's turbinates are permanently enlarged and obstructing airflow, necessitating surgery.

However, this is exactly how surgeons manipulate patients. They exploit the patient's lack of knowledge by presenting a single X-ray image showing swollen turbinates and claiming that surgery is required. If they were genuinely interested in proving that one side's turbinates are consistently enlarged and obstructing airflow, they would need to take multiple X-ray images over time to track nasal airflow throughout the nasal cycle. But since their goal is to operate on as many patients as possible, they deliberately omit this crucial detail. Instead, they use a single image to justify surgery, knowing that most patients are unaware of the nasal cycle.

On the screen, you can see two X-ray images of the same individual taken at different times, showing the nasal cycle shifting from one side to the other. In these images, it is clear that there is sufficient airflow space in both nostrils when the nasal cycle is in a contracted state. This proves that surgery is unnecessary. However, in standard practice, only one image is taken and shown to the patient to convince them that surgery is needed.

Your Body is the Surgeon’s Raw Material for Profit

How You Are Tricked into Turbinate Ablation via Coblation or Radiofrequency Ablation with Devastating Results

Finally, I just want to remind you that surgeons' main goal and purpose in their work is to make a lot of money and perform many surgeries. They enjoy cutting, slicing, and burning, and your body is their raw material, which they process to produce a finished product that results in money flowing into their accounts. Moreover, they are extremely protected by the current legal system, which means they are not even required to tell you what damage you will suffer from their surgery. In fact, they do not even have to fully disclose what they will do during the operation, because afterward, you will not be able to sue them when you are injured and have had parts removed that were not agreed upon.When it comes to nasal surgery, there is no standardized method that surgeons must follow. They have free rein to do whatever they want, as long as they describe their approach as a “proven and scientific method.” The problem is that this is considered a valid method whether the surgeon removes five percent of your lower turbinate or completely removes all three turbinates in each nostril. This means that even if the surgeon performs a procedure that was not agreed upon, you cannot later claim malpractice and win. The surgeon can always argue afterward that an unforeseen necessity arose during the operation, forcing him to expand its scope—for example, that the nasal cavity turned out to be narrower than he initially thought.

If you still doubt the truth of this information and are considering surgery as a solution for your nasal congestion, I urge you to take a few final minutes to read and listen to the stories of patients who have already been harmed. Listen and absorb the suffering they have endured, and take one last moment to consider whether you truly want to have your life irreversibly destroyed by a surgery that could have been completely replaced by natural treatments for nasal congestion.

Empty Nose Syndrome – Bente’s Story: A Life Interrupted by Septoplasty and Turbinate Reduction

Empty Nose Syndrome case · Netherlands

Bente Van de Veerdonk: Empty Nose Syndrome After Septoplasty and Coblation Turbinate Reduction

Bente Van de Veerdonk was a young woman from Oss, Noord-Brabant, whose health deteriorated after nasal surgery in early 2024. The source material documents severe dryness, burning pain, excessive airflow sensation, disturbed breathing, hyperventilation, extreme sleep loss and a relentless search for relief.

Source context: This page preserves the supplied account of Bente’s life, medical history, symptoms and online posts. Statements about the effects of coblation, corticosteroid sprays, Empty Nose Syndrome and the relationship between surgery and later decline are presented as reported in the source material rather than as independent medical findings.
NameBente Van de Veerdonk
BornDecember 28, 1997
LocationOss, Noord-Brabant, Netherlands
StudiesChildcare; Marketing & Communication
Nasal surgerySeptoplasty + bilateral turbinate reduction
Passed awayNovember 2024

Empty Nose Syndrome: A Tragic Story

Bente Van de Veerdonk was a vibrant and determined young woman from Oss, Noord-Brabant, in the Netherlands. Born on December 28, 1997, she had a promising future, balancing aspirations in marketing and communication with a career in childcare.

Her life ended in November 2024, approximately one and a half months before her 27th birthday, after a devastating period of health problems that the source material associates with septoplasty and turbinate reduction.

During the septoplasty, Bente’s inferior turbinates were reduced using coblation. The source describes this as the event that led to Empty Nose Syndrome. Alongside ENS, Bente also lived with Crohn’s disease and thyroid imbalance, adding further complexity to her health.

A Life of Ambition and Dedication

Bente’s educational path reflected her commitment to learning and personal development. She studied childcare at ROC de Leijgraaf from 2014 to 2017 and earned qualifications as a pedagogical employee.

Her dedication to working with children was reflected in her role at Avem Kinderopvang from July 2019 to November 2021, where she worked in care and early childhood education.

She later broadened her professional ambitions by studying marketing and communications through LOI, the Leidse Onderwijsinstellingen, in 2023.

A Multifaceted Health Struggle

In addition to her professional and academic goals, Bente faced chronic health challenges. She had Crohn’s disease, an inflammatory bowel condition, and also experienced thyroid imbalance that left her body feeling fragile and unpredictable.

Despite these challenges, she continued searching for ways to improve her health, including alternative approaches such as homeopathy and consultations with different specialists.

The Nose Surgery That Changed Everything

In early 2024, Bente underwent nasal surgery intended to correct a deviated septum and reduce enlarged turbinates.

The procedure included septoplasty and bilateral turbinate reduction using coblation, a technique commonly presented as a relatively conservative method intended to improve nasal airflow.

According to the source, Bente developed immediate postoperative complications that progressively worsened.

Her postoperative care included corticosteroid nasal sprays. Bente herself believed that these sprays further damaged her nasal mucosa.

Video About Bente Van de Veerdonk’s Case

Severe and Unrelenting Symptoms

Excessive airflow sensationBente felt that too much air was passing through the nose.
Burning nasal painShe described a persistent and severe burning sensation inside the nostrils.
Dryness and crustingSevere nasal dryness and crusting were reported, and she believed steroid spray worsened the problem.
Mouth-open sleepShe slept with her mouth open because of nasal discomfort, which further increased dryness.
Hyperventilation sensationShe described inability to sense airflow during exhalation and said hyperventilation was worst at night.
Extreme sleep deprivationThe source describes periods of as many as five days without restorative sleep.
Constant breathing awarenessShe became intensely focused on breathing and unable to stop monitoring it.
Loss of concentrationEven simple activities such as watching television became difficult.
Systemic symptomsDry eyes, a lump in the throat, painful ears and burning sensations in the chest and limbs were also reported.
Attempts to reduce airflowShe experimented with nose clips, tape and other methods to block excessive airflow, without success.

The source interprets this symptom pattern as consistent with Empty Nose Syndrome, particularly the combination of an excessively open nasal sensation, severe dryness and loss of normal airflow perception.

Other Symptoms Likely Related to ENS

Additional symptoms listed in the source include dry eyes and mouth, difficulty swallowing, a persistent lump-in-the-throat sensation, ear pain and burning across the chest, arms and back.

Other Medical Conditions

The source states that Bente described her thyroid as being “close to being too hard,” which was interpreted there as possible hyperthyroidism or another thyroid disorder. The exact diagnosis is not established by the supplied text.

A Desperate Search for Relief

Bente searched tirelessly for help. She consulted specialists and planned to visit Dr. Dixon, described in the source as an ENS specialist in Rotterdam.

She experimented with nasal ointments, warm drinks and alternative treatments including homeopathy. The relief was described as either brief or nonexistent.

In online forums, she shared her experiences openly and asked other patients for advice, alternating between hope that something might help and despair when the symptoms continued.

“I can’t focus on anything else but my breathing. Sleeping is a BIG issue... I really need help to make it a bit more bearable because now I can’t even focus on the TV. You have to imagine.”

Her posts also described severe insomnia, use of lorazepam, declining mental well-being, isolation and an urgent desire to find a treatment that would make the symptoms bearable.

The Final Months

During the approximately ten months after surgery, the source describes a dramatic deterioration in Bente’s quality of life.

Health problems that had previously been manageable became overwhelming. She endured sleepless nights, chronic pain and a feeling that her body was failing in multiple ways.

Despite continuing to search for treatment and support, her suffering eventually became overwhelming, and she was lost after a final crisis in November 2024.

A Legacy of Awareness

The source material states that Bente was the sixth person known within these ENS support communities to be lost during a severe crisis in 2024.

It also names Charly Audes of France, Tyler Kuckelman of the United States, Océane Flavigny of France, Sam Treffry of Australia and an unnamed 54-year-old Canadian man as other people reported by the same online communities during that year.

The source specifically notes that these reports came from three Facebook ENS support groups with approximately 5,000 to 6,000 members and should not be interpreted as global epidemiological statistics.

The original text argues strongly that Bente’s outcome was directly connected to complications following septal and turbinate surgery and calls for greater medical accountability, stronger warnings and reconsideration of procedures that alter turbinate tissue. That causal and policy position is preserved here as the viewpoint of the source material rather than presented as an independently established conclusion.

Profiles and Supporting Material

Empty Nose Syndrome - Jack Ackland's Silent Struggle: The ENT Surgeon Who Took Down a Combat Pilot

Historical ENS case · U.S. Air Force veteran

Jack Ackland: A WWII Veteran’s Long-Term Suffering After Turbinectomy and Nasal Surgery

Jack Ackland’s story spans military flight training, repeated sinus procedures, chronic pain, disability and a decades-later medical review stating that his post-surgical condition would now be described as Empty Nose Syndrome.

Jack Ackland served in the U.S. military during the World War II era and hoped to continue flying. According to the supplied article and family documentation, worsening sinus problems during flight training led to multiple operations, including turbinate surgery. The procedures were followed by persistent pain, excessive nasal openness, atrophic rhinitis, recurrent respiratory illness and loss of his ability to continue his military flying career.

Historical source note: This page is based on the supplied case material and on excerpts from a 2018 article by Jack’s son, journalist Len Ackland. A later ENT review quoted in that article stated that Jack’s post-surgical condition would today be called “Empty Nose Syndrome.” The supplied material contains conflicting references to the exact year of Jack’s passing, so this page uses “early 1960s” rather than silently choosing one date.
Military roleU.S. Air Force / WWII-era service
Key surgeryTurbinectomy and nasal resection
Later descriptionCondition comparable to ENS
Long-term outcomeChronic pain and disability

Military service, flight training and worsening sinus problems

Jack was a devoted serviceman who wanted to fly. The supplied account states that his sinus symptoms became increasingly difficult during aviation training, where pressure changes intensified facial and sinus pain.

He initially hoped medical treatment would allow him to continue his career. Instead, the case record describes repeated interventions followed by complications that progressively limited his ability to fly and eventually ended that part of his military service.

In a later letter, Jack recalled that the symptoms and pain became so severe during flying that he could no longer continue in the air.

The 1943 nasal surgery and turbinectomy

According to the article excerpts supplied with this page, Jack was treated by Major R. W. Wright, M.D., head of an ENT department. Jack wrote that he had been reluctant to undergo sinus surgery but eventually consented after substantial pressure.

The source material describes removal of nasal cartilage and bone followed by surgery on the turbinates. It states that the ends of the turbinates were resected, the nasal cavity became excessively open and Jack later developed atrophic rhinitis and abnormal airflow.

Submucosal resection The historical account describes removal of cartilage and bone from inside the nose as part of the operation.
Turbinectomy The turbinates were also surgically reduced, with the supplied article later questioning whether too much structure had been removed.
Structural consequences The case narrative describes nasal collapse, enlarged internal space and excessive airflow after surgery.
Atrophic rhinitis This diagnosis appears repeatedly in the historical discussion of Jack’s post-operative condition.

Years of chronic pain, respiratory illness and disability

The supplied records describe a long period of persistent illness after Jack’s operations. He experienced severe headaches, recurrent sore throats, bronchial problems, arthritis attacks, weight loss, weakness and susceptibility to respiratory infections.

A supporting physician, Dr. E. R. Fenton, was quoted as regarding Jack’s disability as permanent and total. The records also describe periods when colleagues saw him leaving work close to collapse and occasions when his wife had to collect him because of the severity of his symptoms.

March 1943
Major nasal surgery during military service

Historical records describe cartilage and bone resection together with turbinectomy.

1943–1946
Persistent symptoms and repeated medical care

The supplied material describes continuing sinus drainage, pain, respiratory complications and reduced physical capacity.

November 1946
Severe hospitalization

Jack was reportedly hospitalized with sinusitis, streptococcal infection and major systemic complications.

1950s
Ongoing disability and VA disputes

Jack continued seeking recognition for the long-term consequences of his sinus and respiratory conditions.

Early 1960s
Final period of severe illness

The family account describes worsening physical suffering, isolation and psychological distress before Jack passed away.

Eleven months later
VA reverses its earlier position

The supplied article states that the VA later recognized a mental disorder directly related to the consequences of Jack’s service-connected sinusitis and bronchitis.

Why Jack Ackland’s case is now discussed as Empty Nose Syndrome

The most important modern connection comes from the retrospective review described by Len Ackland. He asked Dr. Robert M. Meyers, an ENT professor at the University of Illinois in Chicago, to review the details of Jack’s operations and later symptoms.

According to the supplied article, Dr. Meyers explained that nasal surgery in the 1940s was performed much more aggressively than modern practice and stated that Jack’s resulting condition would today be referred to as Empty Nose Syndrome.

That retrospective description is significant because the historical records already described several features now associated with ENS discussions: extensive turbinate loss, an enlarged nasal cavity, excessive airflow, atrophic rhinitis, severe discomfort and profound functional impairment.

The VA appeal and recognition of Jack’s long-term suffering

The supplied case account states that the Department of Veterans Affairs initially declined to connect Jack’s final decline with his service-related disability. His wife appealed that decision while trying to support their children on a modest salary.

Nearly a year later, according to the source material, the VA reversed its earlier position. The agency recognized a marked personality change and psychiatric manifestations associated with an acquired mental disorder, and linked that disorder directly to the consequences of Jack’s sinusitis and bronchitis.

His widow was then granted Dependency and Indemnity Compensation. In the family narrative, that decision represented a delayed acknowledgment of how severe Jack’s chronic physical illness and related psychological distress had become.

Historical records and article excerpts

Historical Jack Ackland medical and military case documents
Historical material associated with the Jack Ackland case. Click to open the original image.
Read selected historical excerpts and findings

Jack’s reluctance before surgery

Jack wrote that he had been determined not to undergo sinus surgery but eventually consented after considerable pressure. The procedure was described as a submucosal operation involving removal of internal nasal cartilage and bone.

Criticism of the operation

The supplied article states that other doctors later questioned the necessity and extent of the surgery. The post-operative picture included an enlarged nasal cavity, atrophic rhinitis and excessive airflow.

Unable to continue flying

Jack later recalled that the pain became worse with flying and that the symptoms were ultimately severe enough to ground him and end his effort to continue an aviation career.

Permanent disability

A physician who had treated Jack for years described persistent posterior sinus drainage, bronchial illness and subsequent arthritis, and regarded his disability as permanent and total.

Later ENT review

Decades later, Dr. Robert M. Meyers reviewed the historical surgical details and, according to Len Ackland’s article, said the condition resulting from those procedures would now be called Empty Nose Syndrome.

Original article and supporting PDF

The original long-form article was written in 2018 by Jack’s son, journalist Len Ackland. It combines family history with military, medical and VA records and later expert review.

The Story of Stéphane Spinhirny: A Cautionary Tale of Rhinoplasty and Empty Nose Syndrome (1982-2013)

Empty Nose Syndrome case · France

Stéphane Spinhirny (1982–2013): Empty Nose Syndrome After Septoplasty and Revision Rhinoseptoplasty

Stéphane Spinhirny documented severe nasal obstruction sensations, dryness, crusting, insomnia and psychological distress after nasal surgery. His blog, videos and case records remain an important first-person account of post-surgical Empty Nose Syndrome.

Stéphane Spinhirny Empty Nose Syndrome after septoplasty and rhinoseptoplasty
Stéphane Spinhirny — original image from the source page.

Stéphane sought nasal surgery for a combination of aesthetic concerns and functional breathing problems associated with a deviated septum. According to the case summary, his first operation took place in October 2011 and was followed immediately by severe new nasal symptoms. A second reconstructive rhinoseptoplasty followed in October 2012.

Source note: This page is based on Stéphane’s own blog, his video testimony, supporting case material and the summary supplied with the original page. Statements about individual surgeons, informed consent, surgical responsibility and the quality of medical care are presented as Stéphane’s reported views and allegations rather than independent findings.
NameStéphane Spinhirny
CountryFrance
Years1982–2013
First surgerySeptoplasty · October 2011
Second surgeryRevision rhinoseptoplasty · October 2012
Main condition reportedEmpty Nose Syndrome

The surgical journey: from septoplasty to severe post-operative symptoms

Stéphane initially hoped surgery would improve both the appearance of his nose and his breathing. The case summary identifies his first procedure as a septoplasty performed in October 2011.

According to the supplied material, septal cartilage was removed during this first operation. Stéphane later described the outcome in extremely negative terms and believed he had not been adequately informed about the potential consequences.

His symptoms reportedly began immediately after the initial procedure. Instead of the improvement he expected, he experienced a persistent sensation of nasal blockage even though the nasal passages were not necessarily physically obstructed.

Empty Nose Syndrome symptoms, insomnia and declining quality of life

Stéphane described a combination of symptoms consistent with the way Empty Nose Syndrome is discussed in patient accounts: paradoxical obstruction, dryness, crusting, abnormal breathing sensation and severe sleep disturbance.

Paradoxical nasal obstruction A constant sensation that the nose was blocked or that breathing was inadequate despite surgical widening.
Dryness and crusting Persistent dryness became a major source of discomfort after the operation.
Severe insomnia Sleep became so difficult that medication was reportedly required.
Psychological distress Chronic breathing discomfort and sleep loss were accompanied by anxiety, frustration and severe low mood.

Stéphane’s writings show how strongly the physical symptoms influenced his emotional state. He repeatedly expressed frustration that medical professionals did not understand or acknowledge the severity of what he was experiencing.

October 2012: reconstructive rhinoseptoplasty

The case summary describes a second operation in October 2012 with Dr. Miriam Bönisch. This procedure is identified as a rhinoseptoplasty intended to reconstruct the nasal septum.

According to the supplied material, cartilage was taken from the ear and used to reconstruct the septum together with a PDS plate. The procedure followed the earlier removal of septal cartilage during the first operation.

October 2011
Initial septoplasty

Septal cartilage was reportedly removed. Severe nasal symptoms began immediately afterward.

2011–2012
Persistent ENS-type symptoms

Stéphane documented obstruction sensations, dryness, crusting, insomnia and major psychological distress.

2012
Complaint to the medical board

The source summary states that Stéphane filed a complaint but did not pursue broader legal action.

October 2012
Revision rhinoseptoplasty

Ear cartilage and a PDS plate were reportedly used in an attempt to reconstruct the septum.

2013
Stéphane passed away

He was 30 years old. His blog and recordings continued to circulate as part of ENS awareness efforts.

Stéphane Spinhirny’s blog: documenting ENS and warning future patients

Stéphane created a blog focused on his experience with septoplasty, rhinoplasty and post-operative complications. His writing was highly critical of the medical care he received and repeatedly urged prospective patients to research surgeons and procedures carefully.

Stéphane Spinhirny blog about septoplasty, rhinoplasty and Empty Nose Syndrome
Screenshot of Stéphane Spinhirny’s blog. Click the image to visit the original blog.
Recurring themes in Stéphane’s writing

His posts focused on informed consent, careful surgeon selection, the relationship between nasal structures and breathing sensation, the difficulty of obtaining help after a poor surgical outcome, and the emotional consequences of persistent insomnia and breathing discomfort.

One of his recurring warnings was that patients should not assume a seemingly limited nasal procedure will necessarily remain limited to the structure discussed before surgery.

Stéphane’s video testimony archive

Stéphane also recorded video testimony about his symptoms and surgical experience. The two original Blogger-hosted videos from the source page are preserved below.

Impact on quality of life and family

The case summary describes Stéphane’s post-operative quality of life as extremely poor. Persistent insomnia and depression were among the later symptoms highlighted in the source material.

He left behind his parents, Denis Spinhirny and Jeannine Poncin. His experience later became part of wider ENS awareness material, particularly discussions about informed consent and the long-term consequences that some patients report after nasal surgery.

Original sources and supporting documentation

Stéphane’s own blog, YouTube account and supporting case PDF are available below.